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脂肪细胞异质性的双稳态调控决定产热
AI summary
中性粒细胞和巨噬细胞来源的 IL-1β 使邻近的棕色脂肪细胞稳定在低 UCP1 状态；寒冷温度通过交感神经减少 IL-1β，促使脂肪细胞转向高 UCP1 状态并增强产热，而较高温度则产生相反效应。
Published by Nature
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