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PRIMPOL 通过重新引发合成重启复制叉的分子基础
AI summary
《Nature》，在线发表：2026年11月11日；doi:10.1038/s41586-026-11276-y。PRIMPOL 通过与复制解旋酶 CDC45–MCM2–7–GINS 相互作用而被招募到停滞的复制叉，促使引物从 PRIMPOL 移交给高保真聚合酶 ε，从而最大限度减少致突变的 DNA 合成。
Published by Nature
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